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Elongation was inhibited by 14.8% at 10⁻⁸ M and by 81.5% at 10⁻⁷ M

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With mouse models not being able to accurately recapitulate human neurodevelopmental periods, another translational step could be developing models of haploinsufficiency within NHPs—something that has already been established for the SHANK3 and MECP2 genes within cynomolgus macaques [35, 36]