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In addition, the high metabolism and proliferation rate of tumor cells activating various mechanisms to obtain the iron required for tumor growth catalyzes the formation of reactive oxygen species (ROS) through Fenton/Haber–Weiss chemistry [10], which can result in both iron-induced cell death (ferroptosis) and the initiation of oncogenic signalling pathways, leading to cellular damage, inflammation, and cancer progression [11,12,13]
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